~11 spots leftby Dec 2027

CAR T-Cell Therapy + Checkpoint Inhibitors for Sarcoma

Recruiting in Palo Alto (17 mi)
MH
SN
NA
Overseen byNabil Ahmed, MD
Age: < 65
Sex: Any
Travel: May Be Covered
Time Reimbursement: Varies
Trial Phase: Phase 1
Recruiting
Sponsor: Baylor College of Medicine
Must not be taking: Immunosuppressants, Corticosteroids
Disqualifiers: HIV, Autoimmune disease, Organ transplant, others
No Placebo Group
Breakthrough Therapy

Trial Summary

What is the purpose of this trial?

The purpose of this study is to learn whether it is safe to give HER2-CAR T cells in combination with an immune checkpoint inhibitor drug (pembrolizumab or nivolumab), to learn what the side effects are, and to see whether this therapy might help patients with sarcoma. Another goal of this study is to study the bacteria found in the stool of patients with sarcoma who are being treated with HER2 CAR T cells and immune checkpoint inhibitor drugs to see if the types of bacteria influence how well the treatment works. The investigators have found from previous research that they can put a new gene into T cells that will make them recognize cancer cells and kill them. They now want to see if they can put a new gene in these cells that will let the T cells recognize and kill sarcoma cells. The new gene that the investigators will put in makes an antibody specific for HER2 (Human Epidermal Growth Factor Receptor 2) that binds to sarcoma cells. In addition, it contains CD28, which stimulated T cells and make them last longer. After this new gene is put into the T cell, the T cell becomes known as a chimeric antigen receptor T cell or CAR T cell. In another clinical study using these CAR T cells targeting HER2 as well as other studies using CAR T cells, investigators found that giving chemotherapy before the T cell infusion can improve the effect the T cells can have. Giving chemotherapy before a T cell infusion is called lymphodepletion since the chemotherapy is specifically chosen to decrease the number of lymphocytes in the body. Decreasing the number of the patient's lymphocytes first should allow the infused T cells to expand in the body, and potentially kill cancer cells more effectively. The chemotherapy used for lymphodepletion is a combination of cyclophosphamide and fludarabine. After the patient receives the lymphodepletion chemotherapy and CAR T cells during treatment on the study, they will receive an antibody drug called an immune checkpoint inhibitor, pembrolizumab or nivolumab. Immune checkpoint inhibitors are drugs that remove the brakes on the immune system to allow it to act against cancer.

Will I have to stop taking my current medications?

The trial protocol does not specify if you need to stop taking your current medications. However, you must be at least 4 weeks from your last cytotoxic chemotherapy and at least 7 days or 3 drug half-lives from your last targeted therapy, and you must have recovered from any acute toxic effects.

What data supports the effectiveness of the treatment CAR T-Cell Therapy + Checkpoint Inhibitors for Sarcoma?

Research shows that fludarabine and cyclophosphamide, when used together, can effectively reduce tumor cells and create a favorable environment for T cell activation, which is crucial for CAR T-cell therapy. Additionally, fludarabine has been shown to improve outcomes in CAR T-cell therapy for leukemia, suggesting potential benefits in similar treatments.12345

Is CAR T-Cell Therapy with Cyclophosphamide and Fludarabine safe for humans?

Cyclophosphamide and fludarabine, used before CAR T-cell therapy, can cause significant reduction in certain immune cells, but they also create a suitable environment for T-cell activation. Some patients may experience side effects like cytokine release syndrome (a severe immune reaction) and neurotoxicity (nerve damage), but these risks can be managed with careful dosing.15678

How is the CAR T-Cell Therapy + Checkpoint Inhibitors treatment for sarcoma different from other treatments?

This treatment is unique because it uses genetically engineered T cells (a type of immune cell) to specifically target and kill cancer cells by recognizing a protein called HER2 on their surface, which is different from traditional chemotherapy that attacks all rapidly dividing cells. Additionally, the combination with checkpoint inhibitors helps to enhance the immune response against the tumor, offering a novel approach for sarcomas where standard treatments are limited.19101112

Research Team

MH

Meenakshi Hegde, MD

Principal Investigator

Baylor College of Medicine

SN

Shoba Navai, MD

Principal Investigator

Baylor College of Medicine

NA

Nabil Ahmed, MD

Principal Investigator

Baylor College of Medicine

Eligibility Criteria

This trial is for young patients aged 1-25 with HER2-positive sarcoma, who have normal heart function and organ health. They must not be pregnant or breastfeeding, agree to use contraception if of childbearing potential, and have no severe allergies to the drugs used in this study. Patients with certain heart conditions, active infections, HIV or tuberculosis are excluded.

Inclusion Criteria

WBC > 2,000/µl
Hgb ≥ 7.0 g/dL (transfusion allowed)
ANC >1,000/ul
See 14 more

Exclusion Criteria

I haven't needed systemic treatment for an autoimmune disease in the last 2 years.
Intercurrent infection
My cancer has formed a large tumor.
See 14 more

Trial Timeline

Screening

Participants are screened for eligibility to participate in the trial

2-4 weeks
1 visit (in-person)

Lymphodepletion Chemotherapy

Participants receive cyclophosphamide for 2 days followed by fludarabine for 5 days to prepare for CAR T cell infusion

1 week
Daily visits (in-person)

CAR T Cell Infusion

Participants receive HER2 CAR T cells intravenously, followed by monitoring for up to 4 hours

1 day
1 visit (in-person)

Checkpoint Inhibitor Treatment

Participants receive pembrolizumab every three weeks or nivolumab every two weeks, starting one week after CAR T cell infusion

6 weeks
Bi-weekly or tri-weekly visits (in-person)

Follow-up

Participants are monitored for safety and effectiveness after treatment, including blood tests and imaging studies

15 years
Regular visits (in-person and virtual)

Treatment Details

Interventions

  • Cyclophosphamide and Fludarabine (Alkylating agents)
  • HER2-CAR T cells (CAR T-cell Therapy)
  • Nivolumab (Checkpoint Inhibitor)
  • Pembrolizumab (Checkpoint Inhibitor)
Trial OverviewThe trial tests a combination of genetically modified T cells (HER2-CAR T cells) and immune checkpoint inhibitors (pembrolizumab or nivolumab). It aims to see if this mix can safely treat advanced sarcoma by enhancing the body's immune response against cancer after pre-treatment with lymphodepletion chemotherapy.
Participant Groups
2Treatment groups
Experimental Treatment
Group I: Arm AExperimental Treatment2 Interventions
autologous HER2 CAR T cells infused in combination with lymphodepletion chemotherapy and the PD-1 antibody pembrolizumab
Group II: Arm 2Experimental Treatment2 Interventions
autologous HER2 CAR T cells infused in combination with lymphodepletion chemotherapy and the PD-1 antibody nivolumab

Find a Clinic Near You

Research Locations NearbySelect from list below to view details:
Texas Children's HospitalHouston, TX
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Who Is Running the Clinical Trial?

Baylor College of Medicine

Lead Sponsor

Trials
1044
Patients Recruited
6,031,000+

National Institutes of Health (NIH)

Collaborator

Trials
2896
Patients Recruited
8,053,000+

Center for Cell and Gene Therapy, Baylor College of Medicine

Collaborator

Trials
114
Patients Recruited
2,900+

The Faris Foundation USA

Collaborator

Trials
1
Patients Recruited
30+

Stand Up To Cancer

Collaborator

Trials
53
Patients Recruited
40,100+

Triumph Over Kid Cancer Foundation

Collaborator

Trials
1
Patients Recruited
30+

St. Baldrick's Foundation

Collaborator

Trials
19
Patients Recruited
9,100+

National Cancer Institute (NCI)

Collaborator

Trials
14080
Patients Recruited
41,180,000+

Findings from Research

Fludarabine modulates composition and function of the T cell pool in patients with chronic lymphocytic leukaemia.Gassner, FJ., Weiss, L., Geisberger, R., et al.[2021]
Fludarabine, cyclophosphamide, and rituximab for the treatment of patients with chronic lymphocytic leukemia or indolent non-Hodgkin lymphoma.Tam, CS., Wolf, M., Prince, HM., et al.[2015]
Combinations of anticancer drugs and immunotherapy.Mitchell, MS.[2015]
Doxorubicin, when used in combination with genetically-modified cancer cell vaccines, showed immunostimulatory effects that enhanced cure rates in established CT26 tumors, suggesting a promising approach for advanced cancer treatment.
In contrast, cyclophosphamide was found to be immunosuppressive and did not improve cure rates when combined with the vaccine, indicating that not all antineoplastic drugs are suitable for combination therapy with cancer vaccines.
Immunomodulatory properties of antineoplastic drugs administered in conjunction with GM-CSF-secreting cancer cell vaccines.Nigam, A., Yacavone, RF., Zahurak, ML., et al.[2020]
Fludarabine exposure predicts outcome after CD19 CAR T-cell therapy in children and young adults with acute leukemia.Dekker, L., Calkoen, FG., Jiang, Y., et al.[2022]
Identifying an optimal fludarabine exposure for improved outcomes after axi-cel therapy for aggressive B-cell non-Hodgkin lymphoma.Scordo, M., Flynn, JR., Gonen, M., et al.[2023]
Immunotherapy of non-Hodgkin's lymphoma with a defined ratio of CD8+ and CD4+ CD19-specific chimeric antigen receptor-modified T cells.Turtle, CJ., Hanafi, LA., Berger, C., et al.[2022]
In a study of 129 patients with relapsed or refractory aggressive B-cell non-Hodgkin lymphoma, different CD19 CAR T-cell products showed significant variations in toxicity and efficacy, with JCAR014 associated with lower severity of cytokine release syndrome and neurotoxicity compared to axicabtagene ciloleucel (axicel).
While JCAR014 and tisagenlecleucel (tisacel) had lower odds of complete response compared to axicel, the study highlights that the type of CAR T-cell product can independently influence treatment outcomes, emphasizing the importance of product selection in therapy.
Impact of CD19 CAR T-cell product type on outcomes in relapsed or refractory aggressive B-NHL.Gauthier, J., Gazeau, N., Hirayama, AV., et al.[2023]
Chimeric antigen receptor T (CAR-T) cell immunotherapy for sarcomas: From mechanisms to potential clinical applications.Thanindratarn, P., Dean, DC., Nelson, SD., et al.[2020]
Adoptive cellular therapy using CAR-modified T cells targeting CD19 has shown significant clinical efficacy in treating relapsed or refractory B-cell acute lymphoblastic leukemia (B-ALL) in both children and adults, with some patients also benefiting from treatment for chronic lymphocytic leukemia (CLL) and B-cell non-Hodgkin lymphoma (B-NHL).
Current research is expanding the use of CAR T-cell therapies to other cancers, including multiple myeloma and solid tumors, while also addressing challenges such as severe cytokine release syndrome and neurologic toxicities associated with the treatment.
Review: Current clinical applications of chimeric antigen receptor (CAR) modified T cells.Geyer, MB., Brentjens, RJ.[2022]
Chimeric Antigen Receptor T cell Therapy and the Immunosuppressive Tumor Microenvironment in Pediatric Sarcoma.Terry, RL., Meyran, D., Fleuren, EDG., et al.[2021]
CAR T cell therapy, which uses genetically engineered T cells to target cancer cells, has shown significant success in treating B cell malignancies by specifically targeting the CD19 antigen.
While CAR T cells have proven effective in hematologic cancers, research is ongoing to expand their use in solid tumors, with various targets like GD2 and HER2 currently under investigation.
Chimeric Antigen Receptor T Cell Based Immunotherapy for Cancer.Li, F., Zhang, T., Cao, L., et al.[2018]

References

Fludarabine modulates composition and function of the T cell pool in patients with chronic lymphocytic leukaemia. [2021]
Fludarabine, cyclophosphamide, and rituximab for the treatment of patients with chronic lymphocytic leukemia or indolent non-Hodgkin lymphoma. [2015]
Combinations of anticancer drugs and immunotherapy. [2015]
Immunomodulatory properties of antineoplastic drugs administered in conjunction with GM-CSF-secreting cancer cell vaccines. [2020]
Fludarabine exposure predicts outcome after CD19 CAR T-cell therapy in children and young adults with acute leukemia. [2022]
Identifying an optimal fludarabine exposure for improved outcomes after axi-cel therapy for aggressive B-cell non-Hodgkin lymphoma. [2023]
Immunotherapy of non-Hodgkin's lymphoma with a defined ratio of CD8+ and CD4+ CD19-specific chimeric antigen receptor-modified T cells. [2022]
Impact of CD19 CAR T-cell product type on outcomes in relapsed or refractory aggressive B-NHL. [2023]
Chimeric antigen receptor T (CAR-T) cell immunotherapy for sarcomas: From mechanisms to potential clinical applications. [2020]
Review: Current clinical applications of chimeric antigen receptor (CAR) modified T cells. [2022]
Chimeric Antigen Receptor T cell Therapy and the Immunosuppressive Tumor Microenvironment in Pediatric Sarcoma. [2021]
12.United Arab Emiratespubmed.ncbi.nlm.nih.gov
Chimeric Antigen Receptor T Cell Based Immunotherapy for Cancer. [2018]